Accelerated tooth movement methods include a combination of orthodontic treatment with a technique that increases alveolar tissue turnover. There are many techniques to enhance tissue turnover (corticotomy, photobiomodulation, shock‐therapy, etc.) but for clinicians who intend to use any of these techniques, it is critically important to understand the accelerated biology of the periodontium that has been therapeutically induced. Doing so requires a re‐visit of the prevailing PDL cell‐mediated tooth movement model in contrast with a spongiosa‐mediated model that fits accelerated orthodontics. In accelerated orthodontic technique, alveolar osteopenia, a transient condition, needs to be sustained long enough for an optimal orthodontic outcome to be achieved. This lecture examines the osteopenic model of tooth movement and how demineralization remineralization of the alveolar osseous structures enable bone matrix transport. Not widely recognized in the orthodontic professional community are the value of the accelerated orthodontic techniques, the scope of implications, and the knowledge base, all of which are examined in this presentation.
Describe the biology associated with accelerated tooth movement
Demonstrate the basic differences in clinical alveolar bone turnover techniques
Compare and contrast two paradigms mediating tooth movement: PDL versus alveolar osteopenia.